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Unknown General 898d1769

In Takayasu infiltrates of mucosa and submucosa of stomach is characteristically seen in Eosinophilic gastroenteritis.

A
Common carotid artery
B
Subclavian artery
C
Renal artery
D
Inferior mesenteric artery
High-Yield Explanation
(Refer: Robbins Pathologic Basis of Disease, 8thedition, pg no: 513-514) Takayasu arteritis (PULSELESS DISEASE) Granulomatous vasculitis of medium and larger arteries. Ocular disturbances. Diminished pulses in the upper extremities. Transmural fibrous thickening of the aorta (aortic arch andgreat vessels). Aortic lesions share many attributes with giant-cell aortitisincluding clinical features and histology; indeed, the distinctionis typically made only on the basis of the age of the patient. In age > 50 years designated as giant-cell aortitis, while thoseunder 50 have Takayasu arteritis. Morphology - Irregular thickening of the vessel wall with intimal hyperplasia Histological appearance Adventitial mononuclear infiltrates. Perivascular cuffing of the vasa vasorum. Mononuclear inflammation in the media. Granulomatous inflammation and Giant cells. Patchy medial necrosis. In late stages, collagenous scarring, with admixed chronic inflammatory infiltrates, occurs in all three layers of the vessel wall. Aortic root involvement causes aortic insufficiency. Frequency of Arteriography Abnormalities in Takayasu’sArteritis

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