Veins not involved in stomach varices
High-Yield Explanation
Gastric varices Gastric varices can develop secondary to poal hypeension, in conjunction with esophageal varices, or secondary to sinistral hypeension from splenic vein thrombosis In generalised poal hypeension, the increased poal pressure is transmitted by the left gastric vein to esophageal varices and by the sho and posterior gastric veins to the fundic plexus and cardiac veins Isolated gastric varices tend to occur secondary to splenic vein thrombosis Splenic blood flows retrograde through the sho and posterior gastric veins into the varices, then hepatomegaly through the coronary vein into the poal vein. Left to right retrograde flow through the gastriepiploic vein to the superior mesenteric vein can explain the development of ectopic varices in the stomach Ref: Sabiston 20th edition Pgno : 1232